Share your story with others:

MedicineNet appreciates your comment. Your comment may be displayed on the site and will always be published anonymously. Patient Comments FAQs

Tell us a bit about your background to make your comments more useful to other MedicineNet users.

Screen Name: *

Gender of Patient:Male Female

Age Range of Patient:

I am a: Patient Caregiver

Enter your Comment

* Screen Name will appear next to the published comment. Please do not include your full name or email address.

By submitting your comment, and other materials (collectively referred to as a "Submission") to MedicineNet, you grant MedicineNet permission to use, copy, transmit, publish, display, edit and modify your Submission in connection with its Web site. MedicineNet will not pay you for your Submission. You represent that you have all rights necessary for MedicineNet to use your Submission as set forth above.

Please keep these guidelines in mind when writing your comment:

  • Please make sure you address the question asked.
  • Due to the overwhelming number of comments received, not all comments will be published.
  • When selecting comments to publish, our staff will choose those that are educational and complement the topic. Please try to stay on topic.
  • Your comment may be edited. We would typically edit comments to make them clearer and more readable. We will remove personal information such as last names, email and web addresses, and other potentially harmful information.
  • We will not notify you if your comment has been published. We suggest that you check back on the topic article regularly.
  • We do not provide medical or healthcare advice, treatment, or diagnosis.

Thank you for participating!

I have read and agree to abide by the MedicineNet Terms and Conditions and the MedicineNet Privacy Policy (required).

To prevent our systems from spam, please complete the following prior to submitting your comment.

What are the types of Ehlers-Danlos syndromes?

Classical type
(formerly types I & II)

Marked joint hypermobility, skin hyperextensibility (laxity), and fragility are characteristic of the classic type of Ehlers-Danlos syndrome. The smooth, velvety skin is fragile and tears or bruises easily with minor trauma. Joint dislocations and scoliosis are common. Joint instability can lead to sprains and strains. This classical type is inherited as an autosomal dominant genetic trait (directly passed on from one parent to child).

Hypermobility type
(formerly type III)

Joint hypermobility is the major manifestation of this form of Ehlers-Danlos syndrome. Any joint can be affected, and dislocations are frequent. This type is also inherited as an autosomal dominant genetic trait.

Vascular type
(formerly type IV, the arterial form)

In this form of Ehlers-Danlos syndrome, spontaneous rupture of arteries and bowel is a serious manifestation that can lead to death. Clubfoot can be present at birth. Skin laxity is of varying degrees. Veins can be very visible through the skin. It is primarily inherited as an autosomal dominant (directly passed on from one parent to child) genetic trait, but recessive (not seen in family members or only in one generation of members of the same family, meaning that an individual must inherit two copies of the mutation, one from each parent) trait inheritance has been described.

Kyphoscoliosis type
(formerly type VI)

Fragile globe of the eyes, significant skin and joint laxity, and severe curvature of the spine (scoliosis) are typical features. Its inheritance pattern is autosomal recessive.

Arthrochalsia type
(formerly type VIIB, arthrochalasis multiplex congenita)

Patients are short in height and severely affected by joint laxity and dislocations. Skin involvement is variable. Both utosomal dominant and recessive inheritance is possible. A skin biopsy can be used to diagnose this disorder.

Dermatosparaxis type
(formerly type VIIC)

Patients have severely fragile skin that is soft and doughy with sagging and folding. This rare form of Ehlers-Danlos syndrome can be diagnosed with a skin biopsy.

Tenascin-X deficient type

Joint hypermobility, hyperelastic skin, and fragile tissue are seen. Patients with this type lack the multiple shrinking (atrophied) scars in the skin that are often seen in classic Ehlers-Danlos. It is inherited as an autosomal recessive genetic trait.

Other rare variant types have been reported in single families.

Return to Ehlers-Danlos Syndrome

See what others are saying

Comment from: Lovezelix, 55-64 Female (Patient) Published: April 21

I have a bad case of Ehlers-Danlos syndrome type 1 severe. At first they said it was type 2 then after blood and skin biopsy it was changed to type 1 severe. I have had many eye surgeries, detachment and strabismus repair in both eyes. I have had bladder prolapse two times, rectal prolapse two times, ovarian and uterine fibroids surgery, total hysterectomy, left shoulder tear repair, right knee arthroscopic surgery and finally total knee replacement. I have had C-4 L4 fusion, abdominal hernias and too many joint dislocations to keep count. I have had hand surgeries and right thumb joint repair. I suffer with severe migraine headache, blurred vision, dizziness, abdominal pain, cramping, very painful bowel movements, and bladder problems. I live with chronic pain level eight, and chronic insomnia due to painful joints and cramping at night.

Was this comment helpful?Yes
Comment from: Teacher5, 55-64 Female (Patient) Published: May 13

When I was a crawling toddler, my legs would bruise very easily. My mom suspected something was different between me and my siblings so she pushed for a diagnosis - Ehlers-Danlos syndrome (EDS). Oddly, my father had some of my same issues, but he never paid it any mind. Whenever we went on vacation, I would end up with stitches or a huge lump wherever I had bumped myself. My skin stretches incredibly, but it"s never been a problem. I have had more cavities than any of my siblings despite trying to take good care of my teeth. My 2 sons have also had their share of stitches. Also, when they each turned 18, even though neither had had any cavities that could be seen by their pediatric dentist, their new dentist diagnosed them with deep cavities, necessitating root canals. Then, their pediatric dentist did a lot of research and called to ask if they had EDS. I was shocked, to say the least. Watch your teeth!

Was this comment helpful?Yes
Comment from: MomWithEDS, 25-34 Female (Patient) Published: November 18

I am a 32 year old mother of two who was diagnosed by with Ehlers-Danlos syndrome (EDS) in May 2013. As a child and teenager it was always thought that I was just very flexible and double-jointed. A few years ago my hips started popping out without warning, and it was extremely painful. After visiting a local orthopedist, she suspected that EDS Hypermobility Type 3 may be my issue. She referred me to another doctor for an official work-up and diagnosis. He was certain I had EDS within minutes of starting the exam. Currently I am doing physical therapy to strengthen my core (hopefully to make my muscles be able to make my joints more stable), but I haven't had any improvement so far that I can tell. Medication wise I take an extended release medicine (Oxycontin) three times a day, along with an anti-inflammatory and another pain medicine (oxycodone IR) for breakthrough pain. This is not the life I had imagined I'd have, but I try to stay positive in the face of pain and accept my situation without letting it own me.

Was this comment helpful?Yes

Health Solutions From Our Sponsors