THURSDAY, April 19 (HealthDay News) -- A heart characteristic linked to sudden cardiac death is more common in athletes than non-athletes, and black male athletes are much more likely to have this characteristic than other athletes, a new study finds.
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The characteristic -- left-ventricular hyper-trabeculation (LVHT) -- is a feature of certain types of cardiomyopathy (chronic disease of the heart muscle). The abnormality is the leading cause of exercise-related sudden cardiac death in athletes.
Researchers looked at 692 athletes in the United Kingdom and found that nearly 7 percent of them had the abnormality, compared with 0.4 percent of non-athletes. And the condition was much more common in black male athletes (about 13 percent) than in other athletes (4 percent).
None of the athletes with left-ventricular hyper-trabeculation met the diagnostic criteria for any form of cardiomyopathy.
The study is slated for presentation April 19 at the World Congress of Cardiology, in Dubai.
"The high prevalence of LVHT among athletes suggests that this may represent part of the spectrum of cardiac adaptations that are known to make up 'athlete's heart'," Dr. Navin Chandra, of St. George's University of London, said in a World Heart Federatio n news release.
Regular athletic training leads to changes in the heart's function and structure. Although many of these changes have been identified, this is the first study to pinpoint left-ventricular hyper-trabeculation as a feature of so-called athlete's heart, according to the release.
"Given that LVHT is a feature of sudden cardiac death, its prevalence among athletes creates greater challenges for doctors trying to differentiate between athlete's heart and a serious medical condition, particularly in black male athletes where the prevalence is much higher," Chandra said.
Because this study was presented at a medical meeting, the data and conclusions should be viewed as preliminary until published in a peer-reviewed journal.
-- Robert Preidt
Copyright © 2012 HealthDay. All rights reserved.
SOURCE: World Heart Federation, news release, April 16, 2012
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